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4F). We then examined if the will increase in pSrc stages in KI platelets final result from Increased Src associations Along with the β

l of entire blood in sodium citrate. The remaining jugular vein was subjected to inject a coagulation Alternative that contains 100 μ

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Even so, below unstimulated situations, the Pro32Pro33 mutation led to elevated Src phosphorylation, facilitated by amplified talin interactions Along with the β

During the current study, we produced a whole new knockin (KI) transgenic mouse model wherever the Pro32Pro33 isoform is expressed from the endogenous integrin β